Haoma Deep Sleep Aid$45.00
Haoma Deep Sleep Aid
$45.00
Haoma Deep Sleep Aid is a non-sedating natural sleep aid formulated with extracts of Espand, an ancestral plant that has been used by our Iranian ancestors for thousands of years in ritualistic traditions. Haoma Deep Sleep Aid is a sub-perceptive microdose formula meant to increase the restorative deep stage of your overnight sleep cycle, helping you form a natural and healthy habit.
“Sleep is the single most effective thing we can do to reset our brain and body health each day – Mother Nature’s best effort yet at contra-death.” – Matthew Walker, author of Why We Sleep
- Haoma Deep Sleep Aid is formulated to contain a precise sub-perceptive microdose of select beta-carbolines to support restorative slow wave deep sleep and brain detoxification.
- Haoma Deep Sleep Aid promotes neuro anti-inflammation, protects the brain from oxidative stress, and supports neurogenesis while you sleep.
- Haoma Deep Sleep Aid is all-natural, non-sedating, non habit-forming, and improves over time.
- Wake up well-rested from a high-efficiency deep sleep.
Product Description
Haoma Deep Sleep Aid is a non-sedating natural sleep aid formulated with extracts of Espand, an ancestral plant that has been used by our Iranian ancestors for thousands of years in ritualistic traditions. Haoma Deep Sleep Aid is a sub-perceptive microdose formula meant to increase the restorative deep sleep stage of your overnight sleep cycle, helping you to form a natural and healthy sleep habit.
These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease
These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease
Instructions
- Take one capsule 15 minutes before sleeping and at least 3 hours after eating.
- Do not exceed the recommended daily dose. Do not take it if you are pregnant or nursing. Consult with your physician prior to use if you have high blood pressure or a medical condition, or if you take any prescription medications (including but not limited to MAO inhibitors and antidepressants).
- Store in a cool, dry place. Keep out of reach of children and pets.
FAQ
How will I feel after taking Haoma Deep Sleep Aid ?
- Haoma Deep Sleep Aid is a sub-perceptive microdose formulation that will not cause you to feel physiological effects. Upon waking the next day, people report feeling refreshed from a good night of deep sleep.
Should I take Haoma Deep Sleep Aid every day?
- The neuromodulatory effects of Haoma Deep Sleep Aid are immediate but sub-perceptive, non-sedative, and not habit-forming. We designed Haoma Deep Sleep Aid’s microdose formulation so that you can take it every night without becoming dependent, a buildup or accumulation.
- Although not habit-forming, we recommend incorporating Haoma Deep Sleep Aid into a consistent nightly ritual in the formation of healthy sleep habits. Over time, your sleep quality and efficiency will continue to improve with the adoption of healthy sleep habits.
Is Magi Sleep Aid safe?
- Haoma Deep Sleep Aid is safe when taken as directed at the recommended dosage.
- Haoma Deep Sleep Aid contains Circadia ®, a purified plant extract of Espand that is formulated to contain a precise microdose of beta-Carbolines. In addition to their anti-inflammatory and neuroprotective properties, beta-Carbolines also function as monoamine oxidase inhibitors (MAOIs) [1] that can cause an increase in blood pressure when consuming foods containing tyramine [2].
- For this reason, Haoma Deep Sleep Aid should only be taken on an empty stomach or at least 3 hours after eating. It’s best to avoid consuming tyramine-rich foods altogether when taking Haoma Deep Sleep Aid .
How does it work?
- Haoma Deep Sleep Aid contains Circadia®, a precise microdose formulation of Espand-derived beta-Carbolines that, while sub-perceptive, begins to affect the central nervous system within 20 minutes of being ingested. The compounds in Circadia® have been shown to:
- Inhibit neurotoxins [3] and provide neuroprotective antioxidant effects [4]
- Inhibit tumor cells [5] and promote the growth of anti-tumor cytokines [6]
- Inhibit inflammatory signaling and the growth of inflammatory prostaglandins [7]
- Promote neurogenesis by increasing BDNF and hNPC levels
- Modulate neuroreceptors such as serotonin, dopamine, acetylcholine, histamine, and imidazoline that are involved in perception and consciousness [8]
- Through these actions, Circadia® promotes deep slow wave sleep, amplifying neuro detox and restorative healing while you sleep
How does Haoma Deep Sleep Aid differ from Mang Lucid Dream Aid?
- Although derived from purified extracts of the same Espand plant, Haoma Deep Sleep Aid contains a different blend and dosage of selected beta-Carbolines than Mang Lucid Dream Aid. Sleep Aid is formulated to be a sub-perceptive microdose, whereas Mang Lucid Dream Aid is formulated to be slightly perceptive mini-dose.
Ancestral Philosophy
Espand – Holy Plant Wisdom
Haoma, also known as Soma, is the sacred potion used ritually since 1600 BCE in Pagan, Zoroastrian, and Vedic traditions of Ancient Persia and India for the pursuit of wisdom, insight, and immortality [9]. Espand, also known as Wild Rue or Peganum Harmala, is believed to be the fundamental ancestral plant used in concocting Haoma and is the key ingredient in Haoma Deep Sleep Aid.
Espand is an abundant source of beta-carbolines [10], a class of naturally occurring organic compounds with psychoactive and neuroprotective properties [11]. Beta-carbolines are found naturally in a few plants, including Espand (P. Harmala) and Yage (Banisteriopsis Caapi), the ancestral Amazonian plant that is the main ingredient in the psychedelic brew Ayahuasca [12]. Thanks to modern science, we now understand the neurochemistry behind what our ancestors discovered thousands of years ago.
Pharmaceutical Science
Beta-Carbolines – Ancestral Secret to Neurological Health
Beta-carbolines such as harmine [13] are unique neuromodulators that have been shown to exhibit anti-inflammatory activity in neurons [14] and promote neurogenesis by increasing levels of brain-derived neurotrophic factor (BDNF) [15] and human neural progenitor cells (hNPC) [16].
Beta-carbolines also interact with the sleep cycle via the pineal gland, the part of the brain that regulates the body’s circadian rhythm. The pineal gland regulates the sleep cycle via the synthesis of melatonin from serotonin (the “happiness hormone”) [17], and is also believed to produce both beta-carbolines and the psychedelic compound DMT (the “spirit molecule”) via alternate pathways [18].
Supplementation with Haoma Deep Sleep Aid has been shown in individuals to increase the % of time spent in the stage of deep sleep per night.
The deep sleep stage is characterized by a prevalence of low-frequency delta brainwaves, often termed slow wave sleep [19]. Deep sleep is particularly important to the functioning of the glymphatic system [20], which flushes and detoxifies the brain of toxins such as amyloid plaques during this slow-wave phase of sleep [21]. Abnormal sleep patterns that result in nonrestorative sleep, restlessness, and fatigue are characterized by a preponderance of mid-frequency alpha brainwaves during sleep cycles [22].
Supplementation with Haoma Deep Sleep Aid has been shown in individuals to increase slow-wave brain patterns during sleep, promoting deep sleep, restoration, and neurological detox.
Brainwave Activity during the 1st Deep Sleep Cycle Onset
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Left: Prior to Haoma Deep Sleep Aid | Right: After One Week Haoma Deep Sleep Aid
Slow wave deep sleep is critical not just for feeling refreshed in the morning but for long-term neurological health.
References
- [1] Mckenna, Dennis J., et al. “Monoamine oxidase inhibitors in South American hallucinogenic plants: Tryptamine and β-carboline constituents of Ayahuasca.” Journal of Ethnopharmacology, vol. 10, no. 2, Apr 1984, pp 195-223. https://doi.org/10.1016/0378-8741(84)90003-5
- [2] McCabe, Beverly J. “Dietary tyramine and other pressor amines in MAOI regimens: A review.” Journal of the American Dietetic Association, vol.86, no. 8, Aug 1986, pp. 1059-1064. https://doi.org/10.1016/S0002-8223(21)04074-8
- [3] Herraiz, Tomás, and Hugo Guillén. “Inhibition of the bioactivation of the neurotoxin MPTP by antioxidants, redox agents and monoamine oxidase inhibitors.” Food and Chemical Toxicology, 2011 Aug;49(8):1773-81. doi: 10.1016/j.fct.2011.04.026.
- [4] Samoylenko, Volodymyr, et al. “Banisteriopsis caapi, a unique combination of MAO inhibitory and antioxidative constituents for the activities relevant to neurodegenerative disorders and Parkinson’s disease.” Journal of Ethnopharmacology, vol. 127, no. 2, Feb 2010, pp. 357-367. https://doi.org/10.1016/j.jep.2009.10.030
- [5] Bikadar, SM, et al. “Cerebroprotective effect of isolated harmine alkaloids extracts of seeds of Peganum harmala L. on sodium nitrite-induced hypoxia and ethanol-induced neurodegeneration in young mice.” Pakistan Journal of Biological Sciences, 01 Dec 2013, 16(23):1687-1697. DOI: 10.3923/pjbs.2013.1687.1697
- [6] Hamsa, T.P., and Girija Kuttan. “Harmine inhibits tumour specific neo-vessel formation by regulating VEGF, MMP, TIMP and pro-inflammatory mediators both in vivo and in vitro.” European Journal of Pharmacology, 2010 Dec 15;649(1-3):64-73. doi: 10.1016/j.ejphar.2010.09.010.
- [7] Moloudizargari, Milad, et al. “Pharmacological and therapeutic effects of Peganum harmala and its main alkaloids.” Pharmacognosy Reviews, 2013 Jul-Dec; 7(14): 199–212. doi: 10.4103/0973-7847.120524
- [8] Kyzar, Evan J., et al. “Psychedelic Drugs in Biomedicine.” Trends in Pharmacological Sciences, vol. 38, no. 11, Nov 2017, pp. 992-1005. https://doi.org/10.1016/j.tips.2017.08.003
- [9] Clark, Matthew. “Soma and Haoma: Ayahuasca analogues from the Late Bronze Age.” Journal of Psychedelic Studies, vol. 3, no. 2, Jun 2019. https://doi.org/10.1556/2054.2019.013
- [10] Airaksinen, MM, and Kari I. “Beta-carbolines, psychoactive compounds in the mammalian body. Part I: Occurrence, origin and metabolism.” Medical Biology. 1981 Feb;59(1):21-34. PMID: 7022042
- [11] Frost, Danielle, et al. “β-Carboline Compounds, Including Harmine, Inhibit DYRK1A and Tau Phosphorylation at Multiple Alzheimer’s Disease-Related Sites.” PLoS ONE 6(5): e19264, May 2011. https://doi.org/10.1371/journal.pone.0019264
- [12] Dennis J McKenna,”Clinical investigations of the therapeutic potential of ayahuasca: rationale and regulatory challenges.” Pharmacology & Therapeutics, Volume 102, Issue 2, 2004, pp. 111-129. https://doi.org/10.1016/j.pharmthera.2004.03.002.
- [13] Berrougui, Hicham, et al. “Protective effects of Peganum harmala L. extract, harmine and harmaline against human low-density lipoprotein oxidation.” Journal of Pharmacy and Pharmacology, Volume 58, Issue 7, July 2006, pp. 967–974, https://doi.org/10.1211/jpp.58.7.0012
- [14] Liu, Xin, et al. “Harmine is an inflammatory inhibitor through the suppression of NF-κB signaling.” Biochemical and Biophysical Research Communications, Jul 2017, pp. 489(3):332-338. doi: 10.1016/j.bbrc.2017.05.126
- [15] Dos Santos, Rafael G., and Jaime E.C. Hallak. “Effects of the Natural β-Carboline Alkaloid Harmine, a Main Constituent of Ayahuasca, in Memory and in the Hippocampus: A Systematic Literature Review of Preclinical Studies.” Journal of Psychoactive Drugs, vol. 49, no. 1, Dec 2016. https://doi.org/10.1080/02791072.2016.1260189
- [16] Dakic Vanja, et al.”Harmine stimulates proliferation of human neural progenitors.” PeerJ, 2016 Dec 6;4:e2727. doi: 10.7717/peerj.2727
- [17] Wu, Ying-Hui, and Dick F. Swaab. “The human pineal gland and melatonin in aging and Alzheimer’s disease.” Journal of Pineal Research, Dec 2004. https://doi.org/10.1111/j.1600-079X.2004.00196.x
- [18] Dean, Jon G., et al. “Biosynthesis and Extracellular Concentrations of N,N-dimethyltryptamine (DMT) in Mammalian Brain.” Scientific Reports, Jun 2019, 9333 https://doi.org/10.1038/s41598-019-45812-w
- [19] “Stages of Sleep” https://www.ncbi.nlm.nih.gov/books/NBK10996/
- [20] “The Sleeping Brain: Harnessing the Power of the Glymphatic System through Lifestyle Choices” https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7698404/
- [21] “Slow wave sleep disruption increases cerebrospinal fluid amyloid-β levels” https://academic.oup.com/brain/article/140/8/2104/3933862
- [22] “Prevalence and Correlates of Alpha-Delta Sleep in Major Depressive Disorders” https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3159543/
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